Key result
Acute myocardial infarction was associated with a higher prevalence of VH-IVUS-derived thin-cap fibroatheroma in non-culprit segments compared to stable angina pectoris (40% vs 24%, p=0.005).
Why the study?
Are non-culprit coronary plaque characteristics assessed by VH-IVUS more vulnerable in patients with AMI compared to those with stable angina pectoris?
Population
70 patients, comprising 38 consecutive patients with acute myocardial infarction and 32 patients with stable…
Comparison
Virtual histology intravascular ultrasound… vs Comparison between AMI and SAP clinical…
Design
Cross-sectional
Authors
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May indicate systemic vulnerability in AMI; hypothesis-generating and requires prospective validation before clinical use.
Cross-Sectional (n=70)
Are non-culprit coronary plaque characteristics assessed by VH-IVUS more vulnerable in patients with AMI compared to those with stable angina pectoris?
Absolute Event Rate: 40% vs 24%
p-value: p=0.005
Patients with acute myocardial infarction exhibit more vulnerable plaque characteristics in non-culprit coronary segments compared to those with stable angina, highlighting systemic plaque instability in acute coronary syndromes.
Tagieva et al. (2014) conducted a cross-sectional in Acute myocardial infarction and stable angina pectoris (n=70). Acute myocardial infarction vs. Stable angina pectoris was evaluated on Prevalence of VH-IVUS-derived thin-cap fibroatheroma (VH-TCFA) in non-culprit segments (p=0.005). Acute myocardial infarction was associated with a higher prevalence of VH-IVUS-derived thin-cap fibroatheroma in non-culprit segments compared to stable angina pectoris (40% vs 24%, p=0.005).
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