Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
December 7, 1998The Journal of Experimental MedicineOpen Access

Accelerated Neutrophil Apoptosis in Mice Lacking A1-a, a Subtype of the bcl-2–related A1 Gene

View Full Paper
Ask AI
Bookmark
Share

Authors

AHAzumi HamasakiTokyo Women's Medical University HospitalFSFujiro SendoYamagata UniversityKNKeiko NakayamaTohoku University

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Hamasaki et al. (1998) studied this question.

synapsesocial.com/papers/6a71b6fc26770c2b8de134a2https://doi.org/10.1084/jem.188.11.1985
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Characterization of A1, a novel hemopoietic-specific early-response gene with sequence similarity to bcl-2 .1993 · 338 citations
  2. 2Bad, a heterodimeric partner for Bcl-xL and Bcl-2, displaces bax and promotes cell death1995 · 2,130 citations
  3. 3Targeted disruption of Bcl-2 alpha beta in mice: occurrence of gray hair, polycystic kidney disease, and lymphocytopenia.1994 · 396 citations
  4. 4Granulocytic differentiation of HL‐60 cells results in spontaneous apoptosis mediated by increased caspase expression1997 · 79 citations
  5. 5Differential expression of Fas (CD95) and Fas ligand on normal human phagocytes: implications for the regulation of apoptosis in neutrophils.1996 · 569 citations