Key result
The KCNQ1 channel displays remarkable flexibility in gating mechanisms, allowing it to function as a voltage-independent channel in epithelial tissues and a voltage-activated channel in cardiac tissues.
Design
Review
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KCNQ1 flexibility clarifies tissue-specific cardiac roles; leaves open therapeutic targeting for future studies.
Key points are not available for this paper at this time.
Liin et al. (2015) reported a review. The KCNQ1 channel displays remarkable flexibility in gating mechanisms, allowing it to function as a voltage-independent channel in epithelial tissues and a voltage-activated channel in cardiac tissues.
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