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August 16, 2004Proceedings of the National Academy of Sciences

Nociceptor-specific gene deletion reveals a major role for Na v 1.7 (PN1) in acute and inflammatory pain

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Authors

MNMohammed A. NassarUniversity of SheffieldLSL Caroline StirlingCorteva (United States)GFGreta ForlaniUniversity of Insubria

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Nassar et al. (2004) studied this question.

synapsesocial.com/papers/6a71d888439bab0cabc3afaahttps://doi.org/10.1073/pnas.0404915101
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Also Consider

Synapse has enriched 2 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Acid‐sensing ion channels ASIC2 and ASIC3 do not contribute to mechanically activated currents in mammalian sensory neurones2004 · 251 citations
  2. 2Identification of PN1, a predominant voltage-dependent sodium channel expressed principally in peripheral neurons1997 · 526 citations