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May 6, 2020Thoracic CancerOpen Access

LncRNA HOTAIR enhances breast cancer radioresistance through facilitating HSPA1A expression via sequestering miR‐449b‐5p

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Authors

SZShuqin ZhangBWBin WangHXHuiwen Xiao

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Zhang et al. (2020) studied this question.

synapsesocial.com/papers/6a7210a7439bab0cabc3c09chttps://doi.org/10.1111/1759-7714.13450
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Deletion of the Inducible 70-kDa Heat Shock Protein Genes in Mice Impairs Cardiac Contractile Function and Calcium Handling Associated With Hypertrophy2006 · 127 citations
  2. 2<i>miR-449a</i> and <i>miR-449b</i> are direct transcriptional targets of E2F1 and negatively regulate pRb–E2F1 activity through a feedback loop by targeting <i>CDK6</i> and <i>CDC25A</i>2009 · 247 citations
  3. 3BRCA1 and Breast Cancer: a Review of the Underlying Mechanisms Resulting in the Tissue-Specific Tumorigenesis in Mutation Carriers2019 · 61 citations
  4. 4LncRNA HOTAIR up-regulation is strongly related with lymph nodes metastasis and LAR subtype of Triple Negative Breast Cancer2019 · 65 citations