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Atypical hemolytic and uremic syndrome (aHUS), as a form of thrombotic microangiopathy, (TMA) is characterized by a genetic or acquired dysregulation of complement activation, leading to endothelial cell lesions and thrombosis of the small vessels, hemolysis, thrombocytopenia, and acute kidney injury.S1 Here, we report 2 cases of complement mediated aHUS triggered by infection with SARS-CoV-2.
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Kaufeld et al. (2021) studied this question.
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