Why the study?
Does CCR2 deficiency reduce infarct size and oxidative stress after myocardial ischemia-reperfusion in mice?
Does CCR2 deficiency reduce infarct size and oxidative stress after myocardial ischemia-reperfusion in mice?
CCR2 deficiency attenuates infarct size and oxidative stress following myocardial ischemia-reperfusion in mice, suggesting the MCP-1/CCR2 pathway as a potential therapeutic target.
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Hypothesis-generating for CCR2 blockade in murine reperfusion injury; human translation remains untested.
Hayasaki et al. (2006) studied this question.
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