Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
January 14, 2004Journal of NeuroscienceOpen Access

5-Aminoimidazole-4-Carboxamide-1-β-4-Ribofuranoside Inhibits Proinflammatory Response in Glial Cells: A Possible Role of AMP-Activated Protein Kinase

View Full Paper
Ask AI
Bookmark
Share

Authors

SGShailendra GiriHenry Ford Health SystemNNNarendra NathBSBrian A. SmithKrishi Vigyan Kendra, Karda

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Giri et al. (2004) studied this question.

synapsesocial.com/papers/6a723c82febe604dd70a8098https://doi.org/10.1523/jneurosci.4288-03.2004
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1THE AMP-ACTIVATED/SNF1 PROTEIN KINASE SUBFAMILY: Metabolic Sensors of the Eukaryotic Cell?1998 · 1,449 citations
  2. 2Induction of cytokine transcripts in the central nervous system and pituitary following peripheral administration of endotoxin to mice1997 · 197 citations
  3. 3The AMP‐activated protein kinase prevents ceramide synthesis de novo and apoptosis in astrocytes2001 · 164 citations
  4. 4AMP-activated protein kinase, a metabolic master switch: possible roles in Type 2 diabetes1999 · 1,004 citations
  5. 5Evidence that AMP triggers phosphorylation as well as direct allosteric activation of rat liver AMP‐activated protein kinase1991 · 225 citations