Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
January 1, 1999International ImmunologyOpen Access

Selective eosinophil transendothelial migration triggered by eotaxin via modulation of Mac-1/ICAM-1 and VLA-4/VCAM-1 interactions

View Full Paper
Ask AI
Bookmark
Share

Authors

GJGui‐Quan JiaGenentechJGJosé-Ángel GonzaloMillennium Engineering and Integration (United States)AHAndrés HidalgoInstituto de Salud Carlos III

Discussion

Loading...

Member takes

Overview

Key Points

Key points are not available for this paper at this time.

Cite This Study

Jia et al. (1999) studied this question.

synapsesocial.com/papers/6a727099febe604dd70a9a8ahttps://doi.org/10.1093/intimm/11.1.1
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Five tumor necrosis factor-inducible cell adhesion mechanisms on the surface of mouse endothelioma cells mediate the binding of leukocytes.1993 · 229 citations
  2. 2Anti-Mac-1 selectively inhibits the mouse and human type three complement receptor.1982 · 605 citations
  3. 3Evidence for a role of the integrin VLA-4 in lympho-hemopoiesis.1991 · 615 citations
  4. 4Role of endothelial-leukocyte adhesion molecule 1 (ELAM-1) in neutrophil-mediated lung injury in rats.1991 · 396 citations
  5. 5Transgenic mice expressing a B cell growth and differentiation factor gene (interleukin 5) develop eosinophilia and autoantibody production.1991 · 300 citations