Rad acts as a novel endogenous regulator of cardiac excitation-contraction coupling and beta-adrenergic signaling by modulating L-type Ca2+ channel activity.
No takes yet. Share an insight, caveat, or question.
Rad modulation of contractility in animal models should not yet change practice; leaves open therapeutic targeting in human heart failure.
Wang et al. (2009) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: