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December 1, 2000Antimicrobial Agents and ChemotherapyOpen Access

High-Level Fluoroquinolone-Resistant Clinical Isolates of Escherichia coli Overproduce Multidrug Efflux Protein AcrA

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Authors

AMAnnarita MazzariolUniversity of VeronaYTYutaka TokueGunma University HospitalTKTiffany M. KanegawaUniversity of Verona

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Mazzariol et al. (2000) studied this question.

synapsesocial.com/papers/6a72c800febe604dd70ac722https://doi.org/10.1128/aac.44.12.3441-3443.2000
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Ineffectiveness of Topoisomerase Mutations in Mediating Clinically Significant Fluoroquinolone Resistance in Escherichia coli in the Absence of the AcrAB Efflux Pump2000 · 215 citations
  2. 2Bacterial Topoisomerases, Anti‐Topoisomerases, and Anti‐Topoisomerase Resistance1998 · 161 citations
  3. 3The local repressor AcrR plays a modulating role in the regulation of acrAB genes of Escherichia coli by global stress signals1996 · 401 citations
  4. 4Association of organic solvent tolerance and fluoroquinolone resistance in clinical isolates of Escherichia coli1998 · 39 citations
  5. 5Quinolone-resistant mutants of escherichia coli DNA topoisomerase IV parC gene1996 · 88 citations