Cell density, but not proliferative status, plays an important role in modulating intracellular Ca2+ dynamics in rat mesangial cells exposed to atriopeptins and NO-generating vasodilators.
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Cell density may modulate mesangial Ca2+ responses in vitro; leaves open relevance to human glomerular disease and requires in vivo confirmation.
Lermioǧlu et al. (1991) studied this question.
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