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April 1, 1994Journal of Biological ChemistryOpen Access

Calponin decreases the rate of cross-bridge cycling and increases maximum force production by smooth muscle myosin in an in vitro motility assay.

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Authors

JHJoe R. HaeberleUniversity of Vermont

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Implication

Calponin may mediate latch-state in vitro; leaves open its physiologic role in human vascular smooth muscle.

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Cite This Study

Joe R. Haeberle (1994) studied this question.

synapsesocial.com/papers/6a73bf3914e943abc869f121https://doi.org/10.1016/s0021-9258(18)99891-1
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Caldesmon inhibits the cooperative turning-on of the smooth muscle heavy meromyosin by tropomyosin-actin1989 · 63 citations
  2. 2Enhanced force generation by smooth muscle myosin in vitro.1994 · 62 citations
  3. 3Smooth and skeletal muscle myosin both exhibit low duty cycles at zero load in vitro1993 · 174 citations
  4. 4PROTEIN MEASUREMENT WITH THE FOLIN PHENOL REAGENT1951 · 318,705 citations