Why the study?
Does maximal versus submaximal resistance exercise improve blood pressure and cardiac autonomic modulation?
Does maximal versus submaximal resistance exercise improve blood pressure and cardiac autonomic modulation?
High-intensity maximal resistance exercise may be safer regarding cardiac autonomic modulation compared to submaximal exercise, as it did not cause increased sympathetic activity during recovery.
Maximal vs submaximal resistance exercise yields comparable post-exercise BP and autonomic responses in healthy youth; leaves open differential effects in hypertensive or older populations.
The aim of the study was verified the blood pressure responses and the cardiac autonomic modulation after the strength exercise in two different conditions (maximal and submaximal). The subjects were divided in three groups, such as: maximal repetitions (age: 20.5 ± 0.6 years, weight: 63.7 ± 14.8, height: 1.7 ± 0.1, body mass index: 22.8 ± 4.5 Kilogram per square meter (kg/m²)), submaximal repetitions (age: 25 ± 4.1 years, weight: 69.1 ± 12.8, height: 1.8 ± 0.1, body mass index: 22.2 ± 1.7 (kg/m²)) and a control group (age: 23.7 ± 3.8 years, weight: 64.2 ± 15, height: 1.7 ± 0.1, body mass index: 21.8 ± 1.9 (kg/m²)). The blood pressure and the Heart Rate R-R intervals were measured before and during one hour after the session, with 10-minutes intervals length between measurements. The analyze of variance did not showed significant differences between experimental protocols to blood pressure (p > 0.05). However, the effect size was able to show that the most intense training caused a reduction in systolic blood pressure at times. Regarding cardiac autonomic response, the group that exercised the submaximal form exhibited a significant increase in LF / HF (p = 0.022) when 20 minutes’ post-exercise. There was a not significant difference in cardiac autonomic modulation between protocols. The high intensity protocol has caused blood pressure reductions in more moments and it was over safer in relation to cardiac autonomic modulation, since it did not cause increased sympathetic activity during recovery.
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Neto et al. (2017) studied this question.
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