Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
November 1, 2000Molecular and Cellular BiologyOpen Access

Cell Signaling Switches HOX-PBX Complexes from Repressors to Activators of Transcription Mediated by Histone Deacetylases and Histone Acetyltransferases

View Full Paper
Ask AI
Bookmark
Share

Authors

MSMaya SalehInstitut National de la Recherche ScientifiqueIRIsabel RambaldiMontreal Neurological Institute and HospitalXYXiang-Jiao YangMcGill University

Discussion

Loading...

Member takes

Overview

Key Points

Key points are not available for this paper at this time.

Cite This Study

Saleh et al. (2000) studied this question.

synapsesocial.com/papers/6a744401f40cdade3da7fa8chttps://doi.org/10.1128/mcb.20.22.8623-8633.2000
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Meis Proteins are Major In Vivo DNA Binding Partners for Wild-Type but Not Chimeric Pbx Proteins1997 · 258 citations
  2. 2<i>Drosophila</i> Hox complex downstream targets and the function of homeotic genes1997 · 179 citations
  3. 3PBX and MEIS as Non-DNA-Binding Partners in Trimeric Complexes with HOX Proteins1999 · 170 citations
  4. 4Meis1 and pKnox1 bind DNA cooperatively with Pbx1 utilizing an interaction surface disrupted in oncoprotein E2a-Pbx11997 · 203 citations
  5. 5The human t(1;19) translocation in pre-B ALL produces multiple nuclear E2A-Pbx1 fusion proteins with differing transforming potentials.1991 · 236 citations