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IN 1956 Morris, Blount and Savage1 described the recovery of a cytopathogenic agent that produced acute respiratory illness in chimpanzees and possibly in human beings. They termed this the chimpanzee coryza agent. In 1957 Chanock and his co-workers2 , 3 reported two isolations of a similar agent from infants with respiratory illness. They also found serologic evidence of infection of a number of additional children from whom it had not been possible to isolate the virus. On the basis of serologic responses, this infection was shown to occur in a significantly greater portion of outpatients with respiratory infections than in those without . . .
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Beem et al. (1960) studied this question.
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