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Complete blood cell count components and their potential role in the pathogenesis of coronary slow-flow phenomenon are discussed in this commentary.
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Supports hypothesis of hematologic involvement in coronary slow-flow; leaves open clinical utility pending prospective validation.
Nasim Arjmand, Mohammad Reza Dehghani Department of Cardiology, Seyyed-al-Shohada Heart Center, Urmia University of Medical Sciences, Urmia, IranDespite the implementation of preventive strategies, ischemic heart disease and stroke remain the main causes of mortality and morbidity worldwide.1,2 Of the cardiovascular diseases, coronary slow-flow phenomenon (CSFP), with a prevalence rate of 1%–7% among patients undergoing diagnostic coronary angiography, has been found to be associated with cardiovascular events, including cardiac arrhythmia and acute coronary syndrome.3–5 However, the potential mechanisms involved in the pathogenesis of CSFP remain unknown. Microvascular and endothelial dysfunctions, inflammation, diffuse atherosclerosis, and increased platelet aggregability have been reported to be the main possible etiologies for CSFP.6,7View original paper by Atlas and colleagues.
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Arjmand et al. (2016) conducted an editorial in Coronary slow-flow phenomenon. Complete blood cell count components was evaluated. Complete blood cell count components and their potential role in the pathogenesis of coronary slow-flow phenomenon are discussed in this commentary.
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