Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
May 25, 2001Journal of the American Chemical Society

Different Mechanisms of Oxidative Stress and Neurotoxicity for Alzheimer‘s Aβ(1−42) and Aβ(25−35)

View Full Paper
Ask AI
Bookmark
Share

Authors

SVS. VaradarajanAndhra UniversityJKJarosław KańskiUniversity of Agriculture in KrakowMAMarina AksenovaUniversity of South Carolina

Discussion

Loading...

Member takes

Overview

Key Points

Key points are not available for this paper at this time.

Cite This Study

Varadarajan et al. (2001) studied this question.

synapsesocial.com/papers/6a75bf0cd17bc701ee161d63https://doi.org/10.1021/ja010452r
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Neurotoxicity of a Fragment of the Amyloid Precursor Associated with Alzheimer's Disease1989 · 913 citations
  2. 2Dramatic Aggregation of Alzheimer Aβ by Cu(II) Is Induced by Conditions Representing Physiological Acidosis1998 · 1,014 citations
  3. 3Chemistry of proximal double bonds: reaction of 9,10-benzotricyclo[4.2.2.22,5]dodeca-3,7,9-triene with bromine1985 · 11 citations
  4. 4Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red.1994 · 1,325 citations
  5. 5The Intracellular Component of Cellular 3‐(4,5‐Dimethylthiazol‐2‐yl)‐2,5‐Diphenyltetrazolium Bromide (MTT) Reduction Is Specifically Inhibited by β‐Amyloid Peptides1995 · 204 citations