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March 8, 2005Biochemical JournalOpen Access

CLOCK is involved in the circadian transactivation of peroxisome-proliferator-activated receptor α (PPARα) in mice

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Authors

KOKatsutaka OishiTokyo University of ScienceHSHidenori ShiraiUniversity of TsukubaNINorio IshidaTokyo Kasei University

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Oishi et al. (2005) studied this question.

synapsesocial.com/papers/6a75eba992ba9b63c6162039https://doi.org/10.1042/bj20041150
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Dexamethasone and Insulin Demonstrate Marked and Opposite Regulation of the Steady‐State mRNA Level of the Peroxisomal Proliferator‐Activated Receptor (PPAR) in Hepatic Cells1994 · 147 citations
  2. 2Altered Patterns of Sleep and Behavioral Adaptability in NPAS2-Deficient Mice2003 · 420 citations
  3. 3Bimodal regulation of mPeriod promoters by CREB-dependent signaling and CLOCK/BMAL1 activity2002 · 550 citations
  4. 4Angiotensin II Induces Circadian Gene Expression of Clock Genes in Cultured Vascular Smooth Muscle Cells2001 · 184 citations