Key result
Myocardial damage may occur in non-culprit lesions due to thrombus migration even following successful treatment of culprit lesions in acute myocardial infarction.
Why the study?
Distal thromboembolism can develop in acute myocardial infarction, but is rare in non-culprit lesions.
Case Report (n=1)
No
This case demonstrates that myocardial damage can occur in non-culprit lesions due to thrombus migration following successful recanalization of the culprit lesion in acute myocardial infarction.
Rare non-culprit embolization possible in AMI; leaves open whether targeted assessment changes management.
Distal thromboembolism can develop in patients with acute myocardial infarction, but is rare in non-culprit lesions. Here, we report a case of acute myocardial infarction in which myocardial damage may have been caused by thrombi originally located in the non-culprit lesion. An 81-year-old woman presented with back pain and was diagnosed with ST-segment elevation acute myocardial infarction. Emergency coronary angiography demonstrated total occlusion in the mid-portion of the left anterior descending coronary artery and severe stenosis in the mid-portion of the right coronary artery. Collateral flow to the right coronary artery from the first septal perforator of the left anterior descending coronary artery, which branched before the total occlusion of the left anterior descending coronary artery, was observed. After recanalization of the left anterior descending coronary by thrombus aspiration, thrombi originally located in the right coronary artery migrated to the distal part. Myocardial imaging with thallium-201 and technetium-99m-pyrophosphate showed damaged but viable myocardium in the inferior wall of the mid-left ventricle.
No takes yet. Share an insight, caveat, or question.
Shirai et al. (2020) conducted a case report in Acute Myocardial Infarction (n=1). Thrombus aspiration and recanalization was evaluated. Myocardial damage may occur in non-culprit lesions due to thrombus migration even following successful treatment of culprit lesions in acute myocardial infarction.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: