Why the study?
Does deendothelializing balloon injury induce nitric oxide synthase expression in arterial smooth muscle cells in a rat model?
Does deendothelializing balloon injury induce nitric oxide synthase expression in arterial smooth muscle cells in a rat model?
Following endothelial injury, arterial smooth muscle cells express inducible nitric oxide synthase, providing a potential mechanism for maintaining arterial patency and nonthrombogenicity.
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Hypothesis-generating for iNOS-mediated patency after arterial injury in rats; leaves open translation to human vascular disease.
Hansson et al. (1994) studied this question.
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