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Introduces a new perspective linking hypoarousal and cognitive constriction to survival in traumatized individuals, suggesting a protective role.
In clinical psychology and traumatology, hypoarousal, emotional numbing, and severe lethargy in Complex Post-Traumatic Stress Disorder (CPTSD) are conventionally classified as debilitating pathological symptoms requiring immediate therapeutic reversal. However, this clinical paradigm fails to account for how severely traumatized individualsundergoing intolerable psychache manage to survive peak suicidal crises without acting upon lethal intent. This paperintroduces the Hypothesis of Lethargic Preservation, arguing that extreme hypoarousal and profound psychomotorretardation act as an involuntary neurobiological emergency brake. By drawing upon Stephen Porges' Polyvagal Theory,Thomas Joiner's Interpersonal Theory of Suicide, and Edwin Shneidman's concept of cognitive constriction, wedemonstrate that dorsal vagal immobilization physically depletes the psychomotor capability required for suicideexecution while rendering both life and death equally void of actionable cognitive momentum. Reconceptualizing lethargynot as mere dysfunction, but as an evolutionary protective circuit, provides profound implications for trauma therapy andoffers a transformative paradigm shift for survivors blaming themselves for immobilizing helplessness.
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Min Jinseong (2026) studied this question.
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