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May 1, 1998Endocrinology

The Thyroid Hormone Receptor Variant α2 Is a Weak Antagonist because It Is Deficient in Interactions with Nuclear Receptor Corepressors*

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Authors

TTTetsuya TagamiKyoto Medical CenterPKPeter KoppUniversity of LausanneWJW. Bruce JohnsonUniversity of Iowa

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Cite This Study

Tagami et al. (1998) studied this question.

synapsesocial.com/papers/6a7748bc576efb0ddaf73a3bhttps://doi.org/10.1210/endo.139.5.6011
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Studies on the repression of basal transcription (silencing) by artificial and natural human thyroid hormone receptor-beta mutants.1995 · 45 citations
  2. 2c-erbA Protooncogenes Mediate Thyroid Hormone-Dependent and Independent Regulation of the Rat Growth Hormone and Prolactin Genes1988 · 132 citations
  3. 3Capacity for cooperative binding of thyroid hormone (T3) receptor dimers defines wild type T3 response elements.1992 · 80 citations
  4. 4Human carboxyl-terminal variant of alpha-type c-erbA inhibits trans-activation by thyroid hormone receptors without binding thyroid hormone.1989 · 129 citations
  5. 5Nuclear Corepressors Enhance the Dominant Negative Activity of Mutant Receptors That Cause Resistance to Thyroid Hormone*1998 · 60 citations