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July 1, 2002Cardiovascular ResearchOpen Access

Dedifferentiation of atrial myocytes during atrial fibrillation: role of fibroblast proliferation in vitro

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Authors

CRCatherine Rücker‐MartinElectrophysiology

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Implication

In vitro atrial myocyte remodeling models warrant mechanistic follow-up; leaves open clinical relevance and therapeutic translation.

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Key points are not available for this paper at this time.

Cite This Study

Catherine Rücker‐Martin (2002) studied this question.

synapsesocial.com/papers/6a77ac4ac5ab23c76cecd022 https://doi.org/10.1016/s0008-6363(02)00338-3

Topics

Left atrial appendage closure Atrial fibrillation Persistent AF management AF ablation outcomes

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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

1Atrophy reversal and cardiocyte redifferentiation in reloaded cat myocardium.1984 · 88 citations
2Influence of the passive anisotropic properties on directional differences in propagation following modification of the sodium conductance in human atrial muscle. A model of reentry based on anisotropic discontinuous propagation.1988 · 336 citations
3Histological alterations in chronically hypoperfused myocardium. Correlation with PET findings.1994 · 298 citations
4Relating extracellular potentials and their derivatives to anisotropic propagation at a microscopic level in human cardiac muscle. Evidence for electrical uncoupling of side-to-side fiber connections with increasing age.1986 · 896 citations