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January 1, 2014ENLIGHTEN (Jurnal Bimbingan dan Konseling Islam)Open Access

Changes in Cardiopulmonary Reserve and Peripheral Arterial Function Concomitantly with Subclinical Inflammation and Oxidative Stress in Patients with Heart Failure with Preserved Ejection Fraction

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Population

32 subjects, comprising 18 patients with heart failure with preserved ejection fraction and 14 healthy…

Design

Case-control

Key result

Patients with HFpEF exhibited significantly decreased peak VO2 (12.0 vs 19.1 mL/min/kg, P<0.001) and elevated inflammatory and oxidative stress biomarkers compared with healthy controls.

Authors

DVDamien VitielloFHFrançois HarelRTRhian M. Touyz

Discussion

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Overview

Inflammation may contribute to HFpEF exercise intolerance; hypothesis-generating and should not yet change practice.

Study Design

Type

Observational (n=32)

Structured PICO

P
Population
32 participants, including 18 patients with heart failure with preserved ejection fraction and 14 healthy controls.
C
Comparator
14 healthy control subjects (CTL)
O
Outcome
Cardiopulmonary reserve (Peak VO2, oxygen uptake efficiency slope), peripheral arterial blood flow, and biomarkers of inflammation and oxidative stress (BNP, CRP, IL-6, TBARS)surrogate

Main Result

Absolute Event Rate: 12% vs 19.1%

p-value: p=<0.001

Patients with HFpEF demonstrate significantly reduced cardiopulmonary reserve and elevated inflammatory and oxidative stress biomarkers compared to healthy controls, despite similar peripheral arterial blood flow.

Cite This Study

Vitiello et al. (2014) conducted an observational in Heart failure with preserved ejection fraction (n=32). Heart failure with preserved ejection fraction vs. Healthy subjects was evaluated on Peak VO2 (mL/min/kg) (p=<0.001). Patients with HFpEF exhibited significantly decreased peak VO2 (12.0 vs 19.1 mL/min/kg, P<0.001) and elevated inflammatory and oxidative stress biomarkers compared with healthy controls.

synapsesocial.com/papers/6a78717c6789de8d0af7c95bhttps://doi.org/10.1155/2014/917271
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