In a canine model of hemorrhagic hypotension, the increase in myocardial oxygen consumption associated with elevated systolic left ventricular pressure is primarily driven by the concomitant increase in dP/dt.
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dP/dt mediates SLVP effects on MVO2 in canine hypotension; extends mechanistic models but leaves open human translation.
Bugge-Asperheim et al. (1970) studied this question.
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