Key result
MyBPC1 physically interacts with MM-CK in a creatine- and pH-dependent manner, acting as an adaptor to connect myosin and MM-CK for efficient energy metabolism.
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Hypothesis-generating for sarcomeric energy transfer; leaves open clinical relevance in HF or cardiomyopathy.
Chen et al. (2011) studied this question. MyBPC1 physically interacts with MM-CK in a creatine- and pH-dependent manner, acting as an adaptor to connect myosin and MM-CK for efficient energy metabolism.
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