This preclinical study challenges clinical assumptions by demonstrating that HL potentials (not LH potentials) may represent the slow pathway, and that the occurrence of accelerated junctional rhythm during ablation may be a coincidental thermal effect rather than a direct marker of slow pathway destruction.
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Questions reliance on LH potentials and AJR for slow pathway ablation; leaves open clinical relevance of HL potentials in humans.
Janse et al. (1996) studied this question.
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