Mutation of the conserved lysine K535 in the kinase homology domain of GC-A reduces its phosphorylation, providing insight into the regulation of natriuretic peptide signaling.
Impaired GC-A activity with K535A mutation; hypothesis-generating for natriuretic peptide regulation but leaves open clinical translation.
No abstract yet. The journal hasn't published one we can show.
No takes yet. Share an insight, caveat, or question.
Edmund et al. (2015) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: