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August 1, 2002Journal of Biological ChemistryOpen Access

Akt Is a Downstream Target of NF-κB

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Authors

FMFanyin MengBeijing Computing CenterLLLi LiuBiotechnology InstitutePCPaul C. ChinThe University of Texas at Dallas

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Meng et al. (2002) studied this question.

synapsesocial.com/papers/6a7bfc5e073f483584dfa8fahttps://doi.org/10.1074/jbc.m112464200
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Activation of Phosphatidylinositol 3-Kinase in Response to Interleukin-1 Leads to Phosphorylation and Activation of the NF-κB p65/RelA Subunit1999 · 651 citations
  2. 2Insulin Resistance and a Diabetes Mellitus-Like Syndrome in Mice Lacking the Protein Kinase Akt2 (PKBβ)2001 · 1,849 citations
  3. 3THE NF-κB AND IκB PROTEINS: New Discoveries and Insights1996 · 5,984 citations
  4. 4Interaction of an NF-kappa B-like factor with a site upstream of the c-myc promoter.1990 · 294 citations
  5. 5Akt Suppresses Apoptosis by Stimulating the Transactivation Potential of the RelA/p65 Subunit of NF-κB2000 · 633 citations