Double target for tumor mass destruction

failure are progressive, it suggests that this counter-regulatory mechanism within the heart is limited and ultimately fails to provide long-term protection.Secondly, we must consider whether or not it will ultimately be beneficial or detrimental to treat (i.e., prevent) cardiac hypertrophy in response to pathologic conditions such as hypertension or valvular disease.In other words, would cardiac decompensation simply occur sooner if one blocks the hypertrophic phase of the response?