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Cycloreversion of the highly mutagenic UV-induced cyclobutane pyrimidine dimer lesion in DNA occurs by a light-induced transfer of an electron through the base stack from a flavin electron donor to the lesion (see schematic representation). This process is even effective when the DNA lesion and flavin donor are separated by an intervening base pair.
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Schwögler et al. (2000) studied this question.
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