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Should not yet change practice; leaves open whether CNS- or stress-targeted therapies reduce cardiovascular events.
There is considerable evidence that the central nervous system is a prime mediator of cardiovascular events. Now that we are able to measure and objectively evaluate the physiologic effects of stress on the cardiovascular apparatus, it should be possible to identify individuals at risk and to offer behavioral and pharmacologic therapies with lifesaving potential.
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Eliot et al. (1983) studied this question.
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