The role of echocardiography in management of hypertrophic cardiomyopathy

Fig. 1. Parasternal long axis and short axis view of an HCM patient with distribution of hypertrophy especially in the septum with MWT of 30 mm

Fig. 2. Patient with septal hypertrophy and MWT of 23 mm (a) with peak gradient of 51 mmHg at rest (b). Echocardiography with injection of contrast in septal branch of the coronary artery with supply of the basal part of the septum (c). Peak gradient of 15 mmHg after ASA (d)

Fig. 3. Longitudinal strain curves from apical four-chamber view in a 53-year-old genotype-positive (MYH7 mutation) relative with normal EF (63%). Average strain from four-chamber view was − 17% (dotted line) and GLS was − 18%, indicating reduced longitudinal function

Fig. 4. Mechanical dispersion by strain echocardiography in two patients with HCM. Horizontal white arrows indicate time to peak strain defined as the time from onset of Q/R to peak negative strain in each segment. Left panel displays longitudinal strain curves and mechanical dispersion in an HCM patient without ventricular arrhythmias. Left panel shows more pronounced mechanical dispersion in an HCM patient with ventricular arrhythmias