There is growing evidence that oxidative modification of low density lipoprotein (LDL), the major cholesterol-carrying lipoprotein in human, plays an important role in the pathogenesis of atheroscler-osis1^. Monocyte-derived macrophages avidly ingest oxidized LDL,but not native LDL, and accumulate large amount of cholesteryl esters, thereby being
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KOHYAMA et al. (1994) studied this question.
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