Why the study?
Understanding how fructose-induced gut microbiota dysbiosis-mediated inflammation influences the pathogenesis of cardiac arrhythmia may provide mechanisms and therapeutic strategies for arrhythmogenesis.
Does excessive intake of fructose promote cardiac arrhythmogenesis through gut dysbiosis and metabolic inflammation?
Design
Narrative review
Authors
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High-fructose intake may promote arrhythmia via gut dysbiosis; hypothesis-generating and requires prospective human trials before practice change.
Does excessive intake of fructose promote cardiac arrhythmogenesis through gut dysbiosis and metabolic inflammation?
Excessive fructose intake may promote cardiac arrhythmogenesis through gut dysbiosis and metabolic inflammation, highlighting the heart-gut axis as a potential therapeutic target.
Cheng et al. (2021) studied this question.
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