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June 1, 1993Journal of Clinical InvestigationOpen Access

Replication, establishment of latency, and induced reactivation of herpes simplex virus gamma 1 34.5 deletion mutants in rodent models.

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Authors

RWRichard J. WhitleyUniversity of Arkansas at Little RockEKEarl R. KernUniversity of Alabama at BirminghamSCS. ChatterjeeGuru Nanak Institutions

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Whitley et al. (1993) studied this question.

synapsesocial.com/papers/6a7ee2d445af3218e19b23f1https://doi.org/10.1172/jci116527
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1The gamma 1(34.5) gene of herpes simplex virus 1 precludes neuroblastoma cells from triggering total shutoff of protein synthesis characteristic of programed cell death in neuronal cells.1992 · 396 citations
  2. 2The herpes simplex virus 1 gene for ICP34.5, which maps in inverted repeats, is conserved in several limited-passage isolates but not in strain 17syn+1990 · 130 citations
  3. 3Recurrent Genital Herpes Simplex Virus Infection in Guinea Pigs1985 · 63 citations
  4. 4Genital Herpes in Guinea Pigs: Pathogenesis of the Primary Infection and Description of Recurrent Disease1982 · 222 citations
  5. 5Inverted Repetitions in the Chromosome of Herpes Simplex Virus1974 · 357 citations