Key result
A 44-year-old woman presenting with acute coronary syndrome was diagnosed with spontaneous coronary artery dissection secondary to fibromuscular dysplasia and successfully managed with medical therapy.
Case Report (n=1)
No
This case highlights the importance of screening for fibromuscular dysplasia in young women presenting with spontaneous coronary artery dissection.
May prompt FMD screening in young women with SCAD; leaves open optimal management strategies.
Spontaneous coronary artery dissection (SCAD) is a well-recognized cause of acute coronary syndrome (ACS) which can lead to myocardial infarction and sudden death. Unlike typical atherosclerosis, SCAD operates through distinct pathophysiology, affecting both individuals with and without conventional cardiovascular risk factors. We present a case of a young female presented with retrosternal chest pain radiating to the left arm, mimicking ACS symptoms with mildly elevated troponin levels, and slightly reduced left ventricular ejection fraction (45%). Subsequent evaluation using coronary angiography unveiled a Type 2A SCAD. A comprehensive computed tomography angiography (CTA) of her entire body revealed findings suggestive of fibromuscular dysplasia (FMD) affecting multiple arteries in different sites. Our case entailed the successful management of a young female patient with SCAD stemming from FMD.
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Al-Mahrizi et al. (2024) conducted a case report in Spontaneous coronary artery dissection (SCAD) and Fibromuscular dysplasia (FMD) (n=1). Fibromuscular dysplasia (FMD) was evaluated on Clinical outcome following medical management. A 44-year-old woman presenting with acute coronary syndrome was diagnosed with spontaneous coronary artery dissection secondary to fibromuscular dysplasia and successfully managed with medical therapy.
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