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January 15, 1998BloodOpen Access

Interferon-α Resistance in a Cutaneous T-Cell Lymphoma Cell Line Is Associated With Lack of STAT1 Expression

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Authors

WSWenn H. SunCPCarlos PabonYAYazan AlsayedNorthwestern University

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Sun et al. (1998) studied this question.

synapsesocial.com/papers/6a7f675ca3e58c8b223000f9https://doi.org/10.1182/blood.v91.2.570
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Mitogen requirements for the in vitro propagation of cutaneous T-cell lymphomas1980 · 422 citations
  2. 2Proteins of transcription factor ISGF-3: one gene encodes the 91-and 84-kDa ISGF-3 proteins that are activated by interferon alpha.1992 · 667 citations
  3. 3Activation of Jak/STAT proteins involved in signal transduction pathway mediated by receptor for interleukin 2 in malignant T lymphocytes derived from cutaneous anaplastic large T-cell lymphoma and Sezary syndrome.1996 · 227 citations
  4. 4Transcriptionally active Stat1 is required for the antiproliferative effects of both interferon alpha and interferon gamma.1996 · 546 citations
  5. 5Role of STAT2 in the Alpha Interferon Signaling Pathway1995 · 281 citations