Why the study?
The pathophysiological mechanisms leading to uraemic cardiomyopathy are not fully understood.
This review proposes that coronary microvascular dysfunction is a key pathophysiological mediator in the development of uraemic cardiomyopathy in chronic kidney disease.
Should not yet change practice in uraemic cardiomyopathy; leaves open whether CMD is causal and a therapeutic target.
The syndrome of uraemic cardiomyopathy, characterised by left ventricular hypertrophy, diffuse fibrosis and systolic and diastolic dysfunction, is common in chronic kidney disease and is associated with an increased risk of cardiovascular morbidity and mortality. The pathophysiological mechanisms leading to uraemic cardiomyopathy are not fully understood. We suggest that coronary microvascular dysfunction may be a key mediator in the development of uraemic cardiomyopathy, a phenomenon that is prevalent in other myocardial diseases that share phenotypical similarities with uraemic cardiomyopathy such as hypertrophic cardiomyopathy and heart failure with preserved ejection fraction. Here, we review the current understanding of uraemic cardiomyopathy, highlight different methods of assessing coronary microvascular function and evaluate the current evidence for coronary microvascular dysfunction in chronic kidney disease.
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Radhakrishnan et al. (2019) studied this question.
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