Why the study?
Complications of atrial fibrillation in myocardial infarction aggravate hemodynamics, warranting further study of vascular elastotonic properties, endothelial function, cardiac remodeling, and anxiety-depressive disorders.
In patients with myocardial infarction, permanent atrial fibrillation is associated with more pronounced structural myocardial changes, vascular stiffness, and anxiety compared to paroxysmal atrial fibrillation.
The widespread prevalence of myocardial infarction and the frequency of complications in the form of atrial fibrillation, makes it relevant to study its pathogenesis and effect on hemodynamics, significantly aggravating the patient’s condition, leading to a deterioration in hemodynamic parameters and the appearance of recurrent myocardial ischemia. At present, a large amount of data has been accumulated on the causes, electrophysiological mechanisms, and hemodynamic consequences of the development of atrial fibrillation during myocardial infarction. This circumstance necessitates both further study of the features of the development and course of atrial fibrillation, and the search for new approaches in organizing and optimizing the treatment of this complication. It is necessary to study in detail the structure of anxiety-depressive disorders, the state of the elastotonic properties of the vascular wall, endothelial function and heart remodeling in patients with myocardial infarction with various forms of atrial fibrillation.
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Neobutov et al. (2020) studied this question.
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