Why the study?
In patients with prior MI, VT may rarely present with a narrower QRS complex, and the underlying mechanisms remain elusive.
Does 3-dimensional activation mapping and targeted catheter ablation eliminate post-infarct ventricular tachycardia masquerading as supraventricular tachycardia in patients with remote anterior myocardial infarction and septal scar?
Does 3-dimensional activation mapping and targeted catheter ablation eliminate post-infarct ventricular tachycardia masquerading as supraventricular tachycardia in patients with remote anterior myocardial infarction and septal scar?
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Post-infarct anteroseptal scar-related VT can present with a narrow QRS mimicking SVT due to proximity of the diastolic pathway and VT exit to the His-Purkinje system, and can be effectively treated with 3D ripple mapping-guided catheter ablation.
Ventricular tachycardia (VT) typically manifests with a broad QRS complex on the surface electrocardiogram (ECG), as the re-entrant circuit involves slow conduction around lines of block within ventricular myocardium.1 VT can manifest deceptive features, such as a sharp intrinsicoid deflection or a narrower QRS, rendering diagnosis challenging.2 Such cases can occur in circuits involving the His-Purkinje system. In patients with prior myocardial infarction (MI), VT may rarely present with a narrower QRS complex, and the underlying mechanisms in such cases remain elusive.
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Chiong et al. (2024) studied this question.
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