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Leaves open clinical translation of inflammation-thrombosis insights; supports targeted trials of novel antithrombotics.
Vessel wall endothelial damage initiates a local inflammatory response, which promotes a prothrombotic state driven by tissue factor, adhesion molecules, and pro-inflammatory cytokines. Understanding how natural inflammatory mechanisms promote a procoagulant state, may lead to the development of new pharmacological interventions targeted at thrombosis.
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A 2005 study studied this question.
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