Population
Mammalian cells transiently expressing mutant alpha1C subunits of L-type Ca2+ channels
Design
Preclinical
Authors
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Highlights potential for targeted Ca channel modulation; leaves open clinical translation from these animal data.
Identifies specific amino acid residues in the IIIS6 and IVS6 segments of L-type calcium channels that form the high-affinity dihydropyridine receptor site.
Peterson et al. (1997) studied this question.
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