Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
April 15, 2000Development

Mouse Gli1 mutants are viable but have defects in SHH signaling in combination with a Gli2 mutation

View Full Paper
Ask AI
Bookmark
Share

Authors

HPH. L. ParkNew York UniversityKPKenneth A. PlattLexicon Pharmaceuticals (United States)MMMichael P. MatiseRutgers, The State University of New Jersey

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Park et al. (2000) studied this question.

synapsesocial.com/papers/6a82ae86e6ac478beb46f705https://doi.org/10.1242/dev.127.8.1593
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Conservation in <i>hedgehog</i> signaling: induction of a chicken <i>patched</i> homolog by <i>Sonic hedgehog</i> in the developing limb1996 · 295 citations
  2. 2Hedgehog Controls Limb Development by Regulating the Activities of Distinct Transcriptional Activator and Repressor Forms of Cubitus interruptus1999 · 379 citations
  3. 3Diminished Sonic hedgehog signaling and lack of floor plate differentiation in <i>Gli2</i> mutant mice1998 · 452 citations
  4. 4Combinatorial <i>Gli</i> gene function in floor plate and neuronal inductions by Sonic hedgehog1998 · 243 citations