Transient thrombocytopenia was induced by ethanol infusion in an alcoholic patient with transient thrombocytopenia. A fall in activity of Cr51 labeled platelets paralleled the fall in platelet count, but no hepatic or splenic sequestration could be demonstrated. It is postulated that ethanol produced a metabolic alteration in the platelets which led to aggregation and temporary sequestration at an unknown site.
No takes yet. Share an insight, caveat, or question.
Post et al. (1968) studied this question.
Synapse has enriched 3 closely related papers on similar clinical questions. Consider them for comparative context: