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S ummary X‐linked anaemia (sla) has been studied in the foetus of the mutant mice of Falconer & Isaacson (1962). The affected male is anaemic by the 19th–20th day of gestation. At this time there is no indication of a defect in the transport of iron to the foetus as indicated by the transfer of 59 Fe and by the content of elementary iron in the total carcass. This suggests that a mechanism other than, or in addition to, intestinal malabsorption is operative in the production of sla.
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Dancis et al. (1970) studied this question.
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