Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
January 15, 2003Genes & DevelopmentOpen Access

Senescence, aging, and malignant transformation mediated by p53 in mice lacking the Brca1 full-length isoform

View Full Paper
Ask AI
Bookmark
Share

Authors

LCLiu CaoShenyang Medical CollegeWLWenmei LiNanjing University of Posts and TelecommunicationsSKSang Soo KimChosun University Hospital

Discussion

Loading...

Member takes

Overview

Key Points

Key points are not available for this paper at this time.

Cite This Study

Cao et al. (2003) studied this question.

synapsesocial.com/papers/6a83cc7f64ff1bb0909965bchttps://doi.org/10.1101/gad.1050003
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 3 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Tumor formation in Brca1 conditional mutant mice2002 · 61 citations
  2. 2Premature senescence involving p53 and p16 is activated in response to constitutive MEK/MAPK mitogenic signaling1998 · 950 citations
  3. 3Brca1 Controls Homology-Directed DNA Repair1999 · 1,249 citations