Traumatic myocardial infarction can lead to severe cardiac remodeling and reduced ventricular function over time, even when coronary arteries appear angiographically normal.
Raises possibility of delayed post-traumatic cardiomyopathy despite normal coronaries; hypothesis-generating and should not yet change practice.
A 23-year-old male with no history of heart disease was admitted to our hospital for an abnormal electrocardiogram of ST-T changes mimicking myocardial infarction. Catheterisation revealed totally normal coronary and peripheral arteries. The echocardiogram and delayed enhancement cardiovascular magnetic resonance imaging indicated a markedly reduced left ventricular function and enlarged left ventricular cavity with evidence of necrotic myocardium. Giving the patient's history of multiple blunt trauma 7 years ago and ‘acute myocardial infarction’ diagnosis at that time, he was diagnosed with traumatic myocardial infarction (TMI). We describe the natural course of such a patient with TMI. Although there is a possibility of spontaneous healing of coronary artery dissection induced by trauma, early revascularisation may be helpful for preventing cardiac remodelling after TMI.
No takes yet. Share an insight, caveat, or question.
Shiwei et al. (2011) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: