Answers are grounded in this paper and cite their sources.
The developmental increases of tyrosine hydroxylase, dopa decarboxylase and doparnine‐β‐hydroxylase activities in neonatal mouse superior cervical ganglion were prevented by administration of the ganglionic blocking agent chlorisondamine. In addition, ganglionic blockade prevented the normal post‐natal increase in ganglion volume and neuron numbers. Inhibition of development followed nicotinic‐receptor blockade, but not muscarinic blockade with atropine.
No takes yet. Share an insight, caveat, or question.
Black et al. (1974) studied this question.
Synapse has enriched 10 related papers. Consider them for comparative context: